Conventional preclinical models often lack the human biology, cellular complexity, and functional endpoints needed to predict clinically relevant neurotoxicity. Animal studies rely on indirect behavioral endpoints, while oversimplified 2D cell models can miss functional nervous system effects that are critical to human safety assessment.

Our human CNS and PNS organoid models close this translational gap by combining human cells, multicellular complexity, functional assessment, and AI neurotoxicology prediction modeling. These integrated capabilities help researchers detect drug-induced changes in neural activity, peripheral nerve conduction, tissue structure, and cell health—supporting more clinically predictive safety assessment.